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Activation of the central melanocortin system chronically reduces body mass without the necessity of long-term caloric restriction
Pharmacological inhibition of mitochondrial -oxidation (e.g., with 4-bromocrotonic acid) or peroxisomal -oxidation (e.g., with thioridazine) under glucose-deprived conditions markedly compromises oligodendrocytes survival and optic nerve conduction, underscoring the central role of this catabolic pathway in white matter energy homeostasis
A recent study from the University of Copenhagen highlights new concerns about GLP-1 medications, including Ozempic, Wegovy, and Mounjaro, focusing on potential bone density loss
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